Asad completed professional studies in Clinical Medicine (LUMHS Pakistan, 2005) and a PhD in Life Sciences (EPFL Switzerland, 2010). Subsequently, he undertook a postdoctoral fellowship at the Department of Pathology and Medicine, University of British Columbia, Canada (2012-2017). In late 2017, he joined Aarhus University (AU) to lead independent research under the auspices of a Marie Curie Fellowship, and in early 2022 have been promoted as an Assistant Professor. During his academic tenures, he has provided scientific leadership and project management in preclinical research applied to neurodegenerative diseases, in particular Alzheimer’s disease (AD) and Parkinson’s disease (PD). Some notable contributions include: i) pathological basis of defective redox homeostasis in PD (PMID: 34092244), ii) perturbed mRNA translation as a common mechanism in AD and PD (PMID: 29961428;PMID: 27752775), and iii) detailed studies on the neuropathological basis of sensorimotor behaviors in animal models of neurodegeneration (PMID: 34136810;PMC7905893). Moreover, his passion for scientific leadership also extends to empowering the research community through methodology development, for example: i) establishing assays for studying neurotoxicity of protein (amyloid) aggregates (PMID: 20539293), ii) somatic gene transfer in rodents using AAV viral vectors (PMC7176396), iii) and guides on analyzing patient-derived gene sets and protein datasets available in public domain for biomarker discovery (PMID: 37414779). In his current research, he employs translational approaches in neurobiology of movement disorders, with focus on novel model development for clinical symptomatology. In parallel, he wants to use these rodent models for the discovery and/or validation of therapeutic molecules for disease modification and/or improvements in clinically relevant sensorimotor phenotypes.